Prohibitin Monoclonal Antibody, 100ul Cell Separation and Collection Mutations in EPHA2 are the

Mutations in EPHA2 are the cause of certain genetically-related cataract disorders

a stimulator of macroautophagy

The Toll-interacting protein regulates inflammatory signaling and is involved in interleukin-1 receptor trafficking and in the turnover of IL1R-associated kinase

It also stimulates the formation and secretion of plasminogen activator by synovial cells

and two MAGE domains in the C-terminus

Prohibitin Monoclonal Antibody, 100ul Cell Separation and Collection Mutations in EPHA2 are theThis gene is evolutionarily conserved, and its product is proposed to play a role in human cellular senescence and tumor suppression. Antiproliferative activity is reported to be localized to the 3' UTR, which is proposed to function as a trans acting regulatory RNA. Several pseudogenes of this gene have been identified. Alternative splicing results in multiple transcript variants.